Search results for "MESH : Muscle"

showing 10 items of 11 documents

Resveratrol: preventing properties against vascular alterations and ageing.

2005

International audience; Cardiovascular diseases are the leading cause of death in developed countries where the common pathological substrate underlying this process is atherosclerosis. Several new concepts have emerged in relation to mechanisms that contribute to the regulation of the vascular diseases and associated inflammatory effects. Recently, potential antioxidants (vitamin E, polyphenols) have received much attention as potential anti-atherosclerotic agents. Among the polyphenols with health benefic properties, resveratrol, a phytoalexin of grape, seem to be a good candidate protecting the vascular walls from oxidation, inflammation, platelet aggregation, and thrombus formation. In …

MESH : Oxidative StressAgingAntioxidantPlatelet AggregationArteriosclerosismedicine.medical_treatmentResveratrolPharmacologymedicine.disease_causeMuscle Smooth Vascularchemistry.chemical_compoundMESH : VasodilationMESH : Foam CellsMESH : Platelet AggregationStilbenesMESH : Cardiovascular DiseasesMESH : Macrophageschemistry.chemical_classificationNeovascularization PathologicPhytoalexinfood and beveragesVasodilationBiochemistryCardiovascular Diseasesmedicine.symptomBiotechnologyLipoproteinsInflammationHealth PromotionMESH : LipoproteinsBiologyMESH : StilbenesMESH : ArteriosclerosismedicineHumans[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyReactive oxygen speciesMechanism (biology)MacrophagesMESH : HumansMESH : Neovascularization PathologicMESH : Muscle Smooth VascularMESH : AgingOxidative StresschemistryAgeingResveratrolMESH : Health PromotionOxidative stressFood ScienceFoam Cells
researchProduct

TRPC1 is regulated by caveolin-1 and is involved in oxidized LDL-induced apoptosis of vascular smooth muscle cells.

2009

International audience; Oxidized low-density lipoprotein (oxLDL) induced-apoptosis of vascular cells may participate in plaque instability and rupture. We have previously shown that vascular smooth muscle cells (VSMC) stably expressing caveolin-1 were more susceptible to oxLDL-induced apoptosis than VSMC expressing lower level of caveolin-1, and this was correlated with enhanced Ca(2+) entry and pro-apoptotic events. In this study, we aimed to identify the molecular events involved in oxLDL-induced Ca(2+) influx and their regulation by the structural protein caveolin-1. In VSMC, transient receptor potential canonical-1 (TRPC1) silencing by ARN interference prevents the Ca(2+) influx and red…

MESH: Lipoproteins LDLVascular smooth muscleOxysterolCaveolin 1ApoptosisBiologyMESH: Base SequenceMESH : RNA Small InterferingMuscle Smooth VascularTRPC1Transient receptor potential channelMESH: RNA Small InterferingMESH : Cells CulturedHumansMESH: Caveolin 1RNA Small InterferingMESH: TRPC Cation ChannelsCells CulturedTRPC Cation ChannelsMESH: HumansBase SequenceMESH : Gene Expression RegulationMESH: ApoptosisMESH : HumansMESH : TRPC Cation ChannelsMESH : Muscle Smooth VascularArticlesCell BiologyMESH: Muscle Smooth VascularActin cytoskeletonMESH: Gene Expression RegulationCell biologyLipoproteins LDLGene Expression RegulationApoptosisCaveolin 1MESH : Caveolin 1Molecular Medicinelipids (amino acids peptides and proteins)MESH : Base SequenceMESH : Lipoproteins LDLHomeostasisMESH : ApoptosisMESH: Cells Cultured
researchProduct

Apelin treatment increases complete Fatty Acid oxidation, mitochondrial oxidative capacity, and biogenesis in muscle of insulin-resistant mice.

2012

Both acute and chronic apelin treatment have been shown to improve insulin sensitivity in mice. However, the effects of apelin on fatty acid oxidation (FAO) during obesity-related insulin resistance have not yet been addressed. Thus, the aim of the current study was to determine the impact of chronic treatment on lipid use, especially in skeletal muscles. High-fat diet (HFD)-induced obese and insulin-resistant mice treated by an apelin injection (0.1 μmol/kg/day i.p.) during 4 weeks had decreased fat mass, glycemia, and plasma levels of triglycerides and were protected from hyperinsulinemia compared with HFD PBS-treated mice. Indirect calorimetry experiments showed that apelin-treated mice…

MESH: Oxidation-Reduction[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismGlucose uptakeAMP-Activated Protein KinasesInbred C57BLMice0302 clinical medicineAMP-activated protein kinaseMESH : Lipid MetabolismHyperinsulinemiaMESH: AnimalsMESH: AMP-Activated Protein KinasesMESH : Muscle SkeletalMESH : Fatty AcidsBeta oxidationMESH: Lipid Metabolism0303 health sciencesMESH: Muscle SkeletalbiologyMESH : Diet High-FatFatty AcidsMESH: Energy MetabolismMESH : AMP-Activated Protein KinasesMESH: Mitochondria MuscleSkeletal3. Good healthApelinMitochondriaMESH: Fatty AcidsMESH : Cyclic AMP-Dependent Protein KinasesMESH: Insulin ResistanceAlimentation et NutritionApelinIntercellular Signaling Peptides and ProteinsMuscleMESH : Insulin ResistanceOxidation-Reductionmedicine.medical_specialtyMESH : Mitochondria Muscle030209 endocrinology & metabolismMESH : Mice Inbred C57BLMESH: Cyclic AMP-Dependent Protein KinasesDiet High-Fat03 medical and health sciencesInsulin resistanceAdipokinesMESH: Mice Inbred C57BLInternal medicineMESH : MiceInternal MedicinemedicineFood and NutritionAnimalsMuscle SkeletalMESH: Intercellular Signaling Peptides and ProteinsMESH: MiceMESH : Intercellular Signaling Peptides and Proteins030304 developmental biologyMESH : Oxidation-ReductionAMPKmedicine.diseaseLipid MetabolismCyclic AMP-Dependent Protein KinasesMitochondria MuscleDietMice Inbred C57BLMESH : Energy Metabolism[SDV.AEN] Life Sciences [q-bio]/Food and NutritionAMP-Activated Protein Kinases;Animals;Cyclic AMP-Dependent Protein Kinases;Diet;High-Fat;Energy Metabolism;Fatty Acids;Insulin Resistance;Intercellular Signaling Peptides and Proteins;Lipid Metabolism;Mice;Inbred C57BL;Mitochondria;Muscle;Skeletal;Oxidation-ReductionHigh-FatMESH: Diet High-FatMetabolismEndocrinologyMitochondrial biogenesisbiology.proteinMESH : AnimalsInsulin ResistanceEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
researchProduct

Muscular performances at the ankle joint in young and elderly men.

2005

The effect of aging on mechanical and electromyographic characteristics of ankle joint muscles was investigated in 11 young (mean age 24 years) and 12 elderly (mean age 77 years) males. Maximal and submaximal isometric voluntary torques were measured during ankle plantarflexion and dorsiflexion. Electromyographic activities of triceps surae and tibialis anterior muscles were recorded. The elderly group developed equal maximal dorsiflexion torques (42 vs 45 N.m, p >.05), but in plantarflexion, the elderly group was weaker (80 vs 132 N.m, p <.001) and presented a decreased twitch amplitude (11 vs 16 N.m) and lower coactivation (8% vs 15%) than that of the young adults. We established a linear…

MaleAgingMESH: Range of Motion ArticularMESH : Ankle Joint[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH : Electric StimulationMESH: Muscle ContractionMESH : Muscle WeaknessMESH : AgedMESH: Evoked Potentials MotorIsometric exerciseElectromyographyMESH : Evoked Potentials Motor0302 clinical medicineMESH: Ankle JointMESH: AgingMESH: Isometric ContractionRange of Motion ArticularYoung adultMESH : Muscle Skeletal10. No inequalityMESH: AgedMESH: Muscle SkeletalMuscle Weaknessmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Muscle WeaknessMESH: Electric StimulationMESH : AdultCoactivationMESH: Legmedicine.anatomical_structureLinear relationshipMESH : ElectromyographyRange of motionMuscle ContractionAdultmedicine.medical_specialtyMESH : MaleMESH: Research Support Non-U.S. GovMESH: ElectromyographyMESH : Isometric Contraction03 medical and health sciencesPhysical medicine and rehabilitationIsometric Contractionmedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansMESH : Research Support Non-U.S. GovMuscle SkeletalAgedLegMESH: HumansElectromyographybusiness.industryMESH : HumansMean ageMESH: Adult030229 sport sciencesEvoked Potentials MotorElectric StimulationMESH : AgingMESH: MaleMESH : Range of Motion ArticularPhysical therapyMESH : Muscle ContractionTibial NerveGeriatrics and GerontologyAnkleMESH : Legbusinesshuman activitiesAnkle Joint030217 neurology & neurosurgery
researchProduct

Stability of pedalling mechanics during a prolonged cycling exercise performed at different cadences.

2005

The aim of this study was to analyse the effect of pedalling rate on the pattern of mechanical torque application and on neuromuscular fatigue during prolonged cycling exercise. Eleven well-trained individuals performed three 1-h pedalling sessions, at 50 rev.min-1, 110 rev.min-1 and a freely chosen cadence, at an intensity corresponding to 65% of their maximal aerobic power. The mechanical torque applied on the right pedal was recorded for 30 s every 5 min while pedalling. Contractile and neural properties of the quadriceps and hamstring muscles were analysed before and immediately after each of the three pedalling sessions. The post-exercise reduction in knee extensors maximal voluntary c…

MaleHamstring muscles[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Muscle ContractionMuscle RelaxationMESH : Analysis of VarianceElectromyographyCohort Studies0302 clinical medicineVoluntary contractionMESH: Risk FactorsRisk FactorsOrthopedics and Sports MedicineMESH: Oxygen ConsumptionMESH : Oxygen ConsumptionMESH: Cohort StudiesKnee extensorsmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]BiomechanicsMESH: Comparative StudyMESH : AdultMESH : Risk FactorsMESH: Muscle FatigueMuscle FatigueMESH : ElectromyographyCyclingCadenceMESH: Physical EnduranceMESH : Physical EnduranceMuscle ContractionAdultmedicine.medical_specialtyMESH: ProbabilityMESH : ProbabilityMESH : MalePhysical ExertionMESH : ExertionMESH : Cohort StudiesPhysical Therapy Sports Therapy and RehabilitationSensitivity and SpecificityMESH : Muscle RelaxationMESH: BicyclingMESH: Electromyography03 medical and health sciencesPhysical medicine and rehabilitationOxygen ConsumptionMESH: Analysis of VariancemedicineMESH: Exertion[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansProbabilityAnalysis of VarianceMESH : SMESH: Humansbusiness.industryElectromyographyMESH : HumansMESH : Comparative StudyMESH: Adult030229 sport sciencesNegative workMESH : Muscle FatigueMESH: MaleBicyclingbody regionsMESH : Exercise TestTorqueMESH : BicyclingMESH: SMESH: Muscle RelaxationPhysical therapyExercise TestPhysical EnduranceMESH : Muscle ContractionbusinessMESH: Exercise Testhuman activities030217 neurology & neurosurgeryJournal of sports sciences
researchProduct

Strength training in old age: adaptation of antagonist muscles at the ankle joint.

2005

The purpose of this study was to determine whether strength training could reduce the deficit in plantarflexion (PF) maximal voluntary contraction (MVC) torque observed in previous studies in older subjects relative to young adults. Accordingly, the effects of a 6-month strength training program on the muscle and neural properties of the major muscle groups around the ankle were examined. PF and dorsiflexion (DF) isometric MVC torques were measured and surface electromyographic activity of the triceps surae and tibialis anterior muscles was recorded. The strength training program was very effective in improving strength in PF (+24.5%), and it thus reduced the DF-to-PF MVC torque ratio; in a…

MaleMESH : Ankle Joint[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH : Electric StimulationPhysiologyMESH: Muscle ContractionMESH : AgedMESH: Physical FitnessIsometric exerciseElectromyographyMESH: Research Support Non-U.S. Gov'tMESH : Research Support Non-U.S. Gov't0302 clinical medicineTriceps surae muscleMESH: Ankle JointMESH : FemaleMESH : Muscle SkeletalMESH : Adaptation PhysiologicalMESH : AlgorithmsMESH: AgedMESH: Muscle SkeletalMESH: Middle Agedmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Electric StimulationMiddle AgedAdaptation PhysiologicalCoactivationmedicine.anatomical_structureData Interpretation StatisticalMESH : ElectromyographyFemalemedicine.symptomMESH : Physical FitnessAlgorithmsMuscle ContractionMuscle contractionmedicine.medical_specialtyWeight LiftingStrength trainingMESH : MaleJoint stabilityMESH: AlgorithmsMESH: Electromyography03 medical and health sciencesCellular and Molecular NeurosciencePhysical medicine and rehabilitationPhysiology (medical)medicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansMESH : Middle AgedMESH : Data Interpretation StatisticalMuscle SkeletalAgedMESH: HumansElectromyographybusiness.industryMESH : Humans030229 sport sciencesMESH: Adaptation PhysiologicalElectric StimulationMESH: MalePhysical FitnessPhysical therapyMESH : Muscle ContractionNeurology (clinical)AnklebusinessMESH: Data Interpretation StatisticalMESH: FemaleAnkle Joint030217 neurology & neurosurgery
researchProduct

The Inactivation Principle: Mathematical Solutions Minimizing the Absolute Work and Biological Implications for the Planning of Arm Movements

2008

An important question in the literature focusing on motor control is to determine which laws drive biological limb movements. This question has prompted numerous investigations analyzing arm movements in both humans and monkeys. Many theories assume that among all possible movements the one actually performed satisfies an optimality criterion. In the framework of optimal control theory, a first approach is to choose a cost function and test whether the proposed model fits with experimental data. A second approach (generally considered as the more difficult) is to infer the cost function from behavioral data. The cost proposed here includes a term called the absolute work of forces, reflecti…

MaleMESH: Range of Motion ArticularMESH : Physical ExertionMESH : MovementOptimality criterion[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Computer scienceMESH: Muscle ContractionMESH: GravitationMESH : Models BiologicalMESH: MovementKinematicsMESH: Postural BalanceMESH : Gravitation0302 clinical medicineNeuroscience/Motor SystemsMESH : FeedbackMESH : BiomechanicsRange of Motion ArticularMESH: ArmMESH : Jointslcsh:QH301-705.5Postural BalanceMESH: Biomechanics0303 health sciencesNeuroscience/Behavioral NeuroscienceEcology[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: FeedbackMESH : AdultBiomechanical PhenomenaMathematical theoryMESH: JointsComputational Theory and MathematicsModeling and SimulationArmResearch ArticleGravitationMuscle ContractionComputer Science/Systems and Control TheoryAdultMESH : MaleMovementPhysical ExertionComputational Biology/Computational NeuroscienceMESH: Psychomotor PerformanceModels BiologicalMESH : ArmFeedbackMESH: Physical Exertion03 medical and health sciencesCellular and Molecular NeuroscienceMESH : Postural BalanceControl theory[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]GeneticsHumansNeuroscience/Theoretical NeuroscienceMolecular BiologyEcology Evolution Behavior and SystematicsSimulation030304 developmental biologyMESH: HumansMESH : HumansWork (physics)MESH: Models BiologicalMotor controlMESH: AdultMESH : Psychomotor PerformanceFunction (mathematics)Optimal controlMESH: MaleTerm (time)MESH : Range of Motion Articularlcsh:Biology (General)MESH : Muscle ContractionJoints030217 neurology & neurosurgeryMathematicsPsychomotor PerformancePLoS Computational Biology
researchProduct

Growth hormone potentiates thyroid hormone effects on post-exercise phosphocreatine recovery in skeletal muscle.

2012

International audience; OBJECTIVE: The aim of the study was to determine the respective impact of thyroxine and growth hormone on in vivo skeletal mitochondrial function assessed via post exercise phosphocreatine recovery. DESIGN: The hind leg muscles of 32 hypophysectomized rats were investigated using (31)P nuclear magnetic resonance spectroscopy at rest and during the recovery period following a non tetanic stimulation of the sciatic nerve. Each rat was supplemented with hydrocortisone and was randomly assigned to one of the 4 groups: the group Hx was maintained in hypopituitarism., the group HxT was treated with 1 μg/100g/day of thyroxine (T4), the group HxG with 0.2 IU/kg/day of recomb…

MalePhosphocreatineThyroid hormonesEndocrinology Diabetes and MetabolismMESH: Random AllocationThyroid GlandSkeletal muscleHypopituitarismMESH: Physical Conditioning AnimalMESH: Drug SynergismNuclear magnetic resonancechemistry.chemical_compoundRandom Allocation0302 clinical medicineEndocrinologyMESH: Human Growth HormoneMESH: AnimalsMESH : Muscle Skeletal0303 health sciencesMESH: Muscle Skeletal[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingMESH : RatsHuman Growth HormoneThyroidDrug Synergismmedicine.anatomical_structuremedicine.drugmedicine.medical_specialtyMESH : Drug SynergismMESH: RatsMESH : MaleSomatotropin030209 endocrinology & metabolismMESH: PhosphocreatinePhosphocreatineMESH : Random Allocation03 medical and health sciencesIn vivoInternal medicine[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyPhysical Conditioning AnimalMESH : Thyroxinemedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingAnimalsHumansMESH : PhosphocreatineMESH : Human Growth HormoneMitochondrionMESH : Physical Conditioning AnimalMuscle Skeletal030304 developmental biologyHydrocortisoneMESH: HumansMESH : HumansSkeletal muscleMESH : Thyroid GlandMESH: Thyroxinemedicine.diseaseMESH: MaleMESH: Thyroid GlandRatsThyroxineEndocrinologychemistryRatMESH : AnimalsTetanic stimulation[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyHormoneGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
researchProduct

Abnormalities of mitochondrial functioning can partly explain the metabolic disorders encountered in sarcopenic gastrocnemius.

2007

International audience; Aging triggers several abnormalities in muscle glycolytic fibers including increased proteolysis, reactive oxygen species (ROS) production and apoptosis. Since the mitochondria are the main site of substrate oxidation, ROS production and programmed cell death, we tried to know whether the cellular disorders encountered in sarcopenia are due to abnormal mitochondrial functioning. Gastrocnemius mitochondria were extracted from adult (6 months) and aged (21 months) male Wistar rats. Respiration parameters, opening of the permeability transition pore and ROS production, with either glutamate (amino acid metabolism) or pyruvate (glucose metabolism) as a respiration substr…

Malemuscle atrophyMESH : Cell Aging[SDV]Life Sciences [q-bio]MESH : Reactive Oxygen SpeciesMitochondrion0302 clinical medicineGlycolysisMESH: AnimalsMESH : Muscle SkeletalMESH : Fatty AcidsCellular SenescencePhospholipidsMESH: Superoxide Dismutasereactive oxygen speciesMESH : Free Radicals0303 health sciencesMESH: Muscle SkeletalMESH : RatsFatty Acidsfatty acid profile of mitochondrial lipidsMESH: Reactive Oxygen SpeciesPyruvate dehydrogenase complexMESH: Fatty Acidsmitochondria[SDV] Life Sciences [q-bio]BiochemistryMESH: Cell AgingMESH: CalciumMESH : MitochondriaCell agingPyruvate decarboxylationmedicine.medical_specialtyFree RadicalsMESH: RatsCellular respirationMESH: MitochondriaMESH : MaleCell Respirationchemistry.chemical_elementOxidative phosphorylationBiologyCalciumMESH : Rats WistarMESH : Phospholipids03 medical and health sciencesMESH: Free RadicalsInternal medicinemedicineAnimalsMESH : Superoxide DismutaseRats WistarMuscle SkeletalMESH : Calcium030304 developmental biologyMESH: Phospholipidscalciumpermeability transition poreSuperoxide Dismutaseagingaging;calcium;fatty acid profile of mitochondrial lipids;mitochondria;muscle atrophy;permeability transition pore;reactive oxygen species;Animals;Calcium;Cell Aging;Cell Respiration;Fatty Acids;Free Radicals;Male;Mitochondria;Muscle;Skeletal;Phospholipids;Rats;Wistar;Reactive Oxygen Species;Superoxide DismutaseCell BiologyMESH: Rats WistarMESH: MaleRatsEndocrinologychemistryMESH : Cell RespirationMESH : AnimalsMESH: Cell Respiration030217 neurology & neurosurgery
researchProduct

Is the beta3-adrenoceptor (ADRB3) a potential target for uterorelaxant drugs?

2007

International audience; The management of premature birth still remains unsatisfactory. Since the relative lack of efficiency and/or safety of current tocolytic agents have been highlighted, it is necessary to develop new uterorelaxant drugs deprived of important maternal and foetal side effects. Our work reported in this review focuses on a potential new target for tocolytic drugs, the beta3-adrenoceptor (ADRB3). This third type of ADRB is shown to be present and functional in human myometrium. We demonstrated that ADRB3 agonists are able to inhibit in-vitro spontaneous contractions of myometrial strips, via a cyclic AMP-mediated pathway. Furthermore, we established that ADRB3 is the predo…

Muscle RelaxationMESH : Receptors Adrenergic beta-3MESH : Adrenergic beta-AgonistsUterusAdrenergic beta-3 Receptor AgonistsMESH: Adrenergic beta-AgonistsPharmacologyUterine contractionUterine Contraction0302 clinical medicineMESH: PregnancyMESH : UterusPregnancyObstetrics and GynaecologyMedicineMESH : FemaleMESH: Obstetric Labor Premature[ SDV.MHEP.GEO ] Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsreproductive and urinary physiology0303 health sciencesMESH : MyometriumMyometriumMESH : Obstetric Labor PrematureObstetrics and GynecologyAdrenergic beta-Agonists3. Good healthmedicine.anatomical_structureMuscle relaxation030220 oncology & carcinogenesisTocolyticMESH: Uterine ContractionMyometriumMESH: MyometriumMESH: UterusFemalemedicine.symptomTocolytic agentmedicine.medical_specialtyAdrenergic receptorAdrenergic beta-3 Receptor Agonists[SDV.MHEP.GEO]Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsMESH : Muscle Relaxation03 medical and health sciencesObstetric Labor PrematureInternal medicineHumans030304 developmental biologyMESH: Humansbusiness.industryMESH : HumansUterus[SDV.MHEP.GEO] Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsMESH : PregnancyEndocrinologyProceedingsReceptors Adrenergic beta-3MESH: Muscle RelaxationbusinessMESH: Receptors Adrenergic beta-3MESH: FemaleMESH : Uterine Contraction
researchProduct